The #1 Organ That Causes High Blood Pressure (and nobody talks about it)

An adult woman was found to be missing her cerebellum

by: Christian Goodman

Anybody diagnosed with high blood pressure will find a volley of ideas and thoughts on what factors and organs are responsible for the spike.

But there is one organ that nobody ever talks about, not even mentions, when discussing high blood pressure. This organ secretly narrows your arteries and raises your blood pressure.

The good news is, when you shed the light on this organ, it’s very easy to tackle it and permanently cure high blood pressure – without medications.

A study conducted by researchers from The School of Medicine at University of Maryland, and Ottawa Heart Institute, found an interesting link between our brains and high blood pressure.

 

In this study, the researchers identified a steroid compound called ouabain, which the brain releases to constrict or dilate the diameter of the arteries throughout the body.

The researchers performed certain experiments on animal models to find this

definite link between the release of ouabain in the bloodstream and constriction of arteries.

Obviously, if the brain releases more ouabain, which narrows the arteries, the blood pressure rises. Just like there is more pressure drinking milkshake through a narrow straw than wide straw.

The most interesting part of this finding is that now we can see our brain has almost complete control over our blood pressure. If we have high blood pressure, it’s because our brain has consciously decided to release ouabain to raise our blood pressure.

So why does the brain make that decision?

The answer is any kind of stress. It can be sensory (traffic noise), emotional (divorce), mental (work task), or physical (workout, disease).

Most of the time this is temporary stress and your brain raises your blood pressure to deal with an immediate threat or condition (this is healthy).

But at some point it becomes chronic. It’s like some neural path gets stuck in this stress circle and can’t get out of it.

Obviously, then, the solution for high blood pressure must be to get us out of this circle. Tell the brain to chill out and stop overdosing on ouabain.

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Anti-Anxiety and Sleeping Pills Increase Risk of Death, New Study Reports

 
A new report says patients who take drugs like Xanax, Valium, and Ambien have a higher risk of dying. What does this mean for the 40 million U.S. adults suffering from anxiety disorders?
Anti-anxiety drugs and sleeping pills significantly increase your chance of dying, according to new research from the UK published in the BMJ.The findings are especially important for the U.S., where anxiety disorders (like panic disorder or social phobia)—the most common mental illness in the country, affecting 40 million adults, or 18 percent of the population—are increasingly treated with prescription drugs.For seven and a half years, researchers there followed 34,727 patients in primary care who were prescribed anxiolytic drugs like Xanax, Valium and Klonopin, and sleep aids like Ambien and Lunesta. They found more than 3.3 times as many patients, prescribed anti-anxiety or sleeping pills, died in the follow-up period. Hazard ratios were largest for benzodiazepines—the most commonly prescribed drug class.

“Researchers found more than 3.3 times as many patients, prescribed anti-anxiety or sleeping pills, died in the follow-up period.”

Researchers controlled for factors linked with early deaths including age, smoking and drinking habits, other prescriptions and socioeconomic status, and most importantly, sleep disorders and anxiety itself.

The BMJ study is just the latest in a line of research suggesting that these drugs can have dangerous, possibly permanent side effects. Not only are they addictive, studies have shown they are associated with cognitive and psychomotor impairments, falls, and unintentional injuries. 

“That’s not to say that they cannot be effective,” Scott Weich, Professor of Psychiatry at the University of Warwick said in the study’s release. “But particularly due to their addictive potential we need to make sure that we help patients to spend as little time on them as possible and that we consider other options, such as cognitive behavioral therapy, to help them to overcome anxiety or sleep problems.”

Use of anti-anxiety drugs can also quickly build a tolerance—in as little as two weeks—that renders them ineffective.

Vitamin D needed by women to maintain cognitive health

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The October, 2012 issue of theJournals of Gerontology Series A: Biological Sciences and Medical Sciences published the outcome of a study conducted by Yelena Slinin, MD, MS, of Minneapolis’ Veterans Administration Medical Center which uncovered a protective effect for higher vitamin D levels against the development of cognitive decline in women.

The study included 6,257 women enrolled in the Study of Osteoporotic Fractures. Serum 25-hydroxyvitamin D was measured upon enrollment and tests of cognitive function were administered at the beginning of the study and at follow-up after four years. Women with low vitamin D levels of less than 10 nanograms per milliliter (ng/mL) had a 60 percent greater risk of cognitive impairment at the beginning of the study and a 58 percent greater risk of becoming cognitively impaired over follow-up in comparison with those whose levels were at least 30 ng/mL.

In another study published in the November, 2012 issue of the same journal, Cedric Annweiler, MD, PhD, of Angers University Hospital in France and his associates report an association between reduced vitamin D intake and a greater risk of developing Alzheimer’s disease. The study included 498 older women enrolled in the EPIDemiology of OSteoporosis Toulouse cohort study. Dietary questionnaires administered at the beginning of the study were analyzed for the intake of vitamin D from food sources. Over a seven year period, 70 participants developed Alzheimer’s disease. In comparison with those who did not develop dementia or developed other types of dementia, women who developed Alzheimer’s disease consumed less vitamin D. When participants were grouped according to vitamin D intake, those in the top one-fifth were found to have a 77 percent lower risk of Alzheimer’s compared to the lowest fifth.

These studies, and others, reinforce the importance of vitamin D in the maintenance of cognitive health over the course of a lifetime.

Decreased vitamin D levels linked to panic, depression

Decreased vitamin D levels linked to panic, depression

Tuesday, February 12, 2013. In an article that appeared online on January 21, 2013 in the journal Clinical Nutrition, researchers at London’s UCL Institute of Child Health report an association between reduced vitamin D levels and an increased risk of panic disorder and depression.

The study included 5,966 men and women enrolled in the 1958 British birth cohort, which consisted of 18,558 individuals born in March, 1958 who were followed to the age of 50 years. Questionnaires administered when the participants were 45 years of age obtained information on such vitamin-D related lifestyle factors as the intake of oily fish and vitamin D supplements, time spent outside, body mass index and other factors. Blood samples collected during the same year were analyzed for 25-hydroxyvitamin D levels, and interviews ascertained the presence of common medical disorders including depression, anxiety, panic and phobias. Additional questionnaires administered at 50 years of age identified and scored depressive symptoms.

Having a sufficient vitamin D level of at least 75 nanomoles per liter (nmol/L) at the age of 45 was associated with a 43 percent lower adjusted risk of depression and a 67 percent lower risk of having panic disorder in comparison with subjects whose levels were less than 25 nmol/L. When depressive symptoms at age 50 were examined, 25-hydroxyvitamin D levels ranging from 50 to 85 nmol/L were associated with the lowest risk of depression after adjustment for several factors. Jane Maddock and her coauthors remark that the association between higher vitamin D levels and reduced depression risk may be due to the vitamin’s effects on serotonin or other neurotransmitters.

“The high burden of mental and behavioral disorders and concurrent high prevalence of vitamin D insufficiency worldwide highlight the potential importance of our findings,” the authors write. “Our results suggest that low 25(OH)D is associated with higher prevalence of depression and panic and that 25(OH)D is modestly and non-linearly associated with subsequent depressive symptoms.”

“Further prospective and experimental work is required to replicate these findings, clarify causality and establish the most effective 25(OH)D status for maximum benefit” they conclude.